Fig. 8From: Glutaminolysis regulates endometrial fibrosis in intrauterine adhesion via modulating mitochondrial functionSchematic diagram of glutaminolysis in endometrial fibrosis. After damage, GLS1-mediated glutaminolysis is activated in endometrial stromal cells, causing a stroma-myofibroblast transition via mTOR pathway. Glutaminolysis participated in energy supply and maintenance of mitochondrial function during ESCs activationBack to article page