Fig. 2From: Prenatal ethanol exposure and changes in fetal neuroendocrine metabolic programmingThe intrauterine programing mechanism of adult metabolic diseases induced by PEE. GC, glucocorticoid; IGF1, insulin like growth factor 1; IUGR, intrauterine growth retardation; 11β-HSD2, 11β-hydroxysteroid dehydrogenase 2; P-gp, P-glycoprotein; HPA axis, hypothalamic-pituitary-adrenal axisBack to article page