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Fig. 5 | Biological Research

Fig. 5

From: Neuronal GRK2 regulates microglial activation and contributes to electroacupuncture analgesia on inflammatory pain in mice

Fig. 5

Schematic representation of the working model. CFA decreased the expression of GRK2 and induced microglial activation and neuroinflammation in the spinal cord and finally induced the development of pain. In contrast, EA regained the GRK2 probably by increasing neuronal GRK2, and then suppressing microglial activation and reducing neuroinflammation which promoted pain relief

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Biological Research

ISSN: 0717-6287

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